COVID’S LONG MARCH: 1918 - 2013
How The U.S. And China Co-Created COVID-19
by William Thomas
In the winter of 1918, coughing and sneezing young men closely-confined among 50,000 fellow-inductees at Fort Riley, Kansas eventually crowd into trains and disembark on the Atlantic seaboard, where they cram into troop transports bound for the slaughter in Europe - where the resulting coronavirus outbreak nearly cancels the concluding battle. Deadlier than the bubonic plague of the Middle Ages, increasingly virulent waves of misnamed “Spanish Flu” outlast the war. Within nine months, as many as 40 million people worldwide are dead.
U.S. germ experiments commence with the Tuskegee Syphilis Study, which secretly infects, examines and refuses to treat 200 black Americans in 1932.
In 1948, lab workers at Camp Detrick in Maryland contract a “terrifyingly infectious” strain of weaponized Acute Brucellosis, despite being vaccinated and wearing rubberised suits.
In 1971, hundreds of thousands of pigs in western Cuba are sacrificed to stop a porker plague, resulting in severely curtailed protein intake for more than 4 million people. Cuba blames the U.S. for its first-ever outbreak of African Swine Fever.
“Virologists and public health officials with the appropriate sophistication were quickly aware that a laboratory release was the most likely origin” of the 1977 H1N1 pandemic, writes Martin Furmanski of the Scientist’s Working Group on Chemical and Biologic Weapons and the Center for Arms Control and Nonproliferation. The genetic blueprint of the strain that had disappeared in 1957 had not changed, as it should have over the intervening decades.
During Sen. Don Riegle’s Senate Hearings that year, army witnesses disclose that from 1949 to 1969 at least 239 “Open-Air” biowarfare tests were conducted in populated areas throughout the United States. Some affected cities included San Francisco, Washington DC, Key West, Panama City, Minneapolis and St. Louis. Civilian casualties resulted.
1980s
After Nicaragua endures a massive outbreak of dengue fever in 1985 and 1986, Fort Detrick researchers admit that vast colonies of mosquitoes infected with the dengue virus and yellow fever viruses are maintained there – as well as hordes of cholera and anthrax-carrying flies, plus swarms of ticks equipped with Colorado and relapsing fevers, Hank P. Albarelli Jr. and Zoe Martell report.
Since the 1980s, the United States Army Medical Research Institute of Infectious Diseases (USAMRIID) based at Fort Detrick, Maryland, has worked closely with virology labs in Wuhan, China.
1996 Thrips bio-attack on Cuba map
1990s
On October 21, 1996, at 10:08 hours, a Cuban Airlines crew reports a single-engine airplane repeatedly laying down spray some 30 kilometres south of Varadero, in Matanzas Province. Based at Patrick Air Force Base, the dual-sprayer-equipped SAR aircraft, N3093M, is operated by the State Department.
On December 18, 1996, the first signs of Thrips plague appear in potato plantations in Matanzas. Until that moment Thrips palmi karay was exotic to Cuban territory. American bio-attacks against the hated Cuban people continue.
2001 - 2009
On Dec. 12, 2001, the Baltimore Sun exposes “CIA contractor Battelle Memorial Institute of West Jefferson, Ohio,” and the “Army at Dugway in Utah” for developing weapons-grade anthrax “identical to the powdery spores used in the mail attacks that have killed five people” in October 2011.
“Dugway is the only facility known in recent years to have processed anthrax spores into the powdery form that is most easily inhaled,” the Washington Post confirms. “Army officials in Washington said yesterday that Fort Detrick does not have the equipment for making dried anthrax spores.”
On Oct. 8, 2002, the U.S. admits it secretly tested chemical and biological weapons on American, Canadians and British populations. In the 1960s and ’70s, Project 112 saw more than two- dozen biological and chemical tests released nerve agents in Alaska and at Dugway in Utah and sprayed bacteria over Hawaii, Maryland and Florida – as well as Canada and Britain, in cooperation with all three governments. Nerve agents released on uninformed test subjects included sarin gas and VX of Gulf War infamy, Bacillus globigii, tabun and soman.
In 2009, Fort Detrick’s germ warfare experiment are suspended after the Pentagon finds “discrepancies” in its inventory of infectious germs during the same year of the latest H1N1 swine flu outbreak. Breaking out again in 2009-2010 as “Swine flu,” H1N1 causes 3,000 to 200,000 deaths. This discrepency is not explained.
Peter Daszak, president of the EcoHealth Alliance, is the lead investigator named on multiple U.S. grants underwriting a low-safety-level, BL-2 lab in Wuhan, China. Daszak co-authors numerous papers with “Bat Woman” Shi Zhengli, including their joint 2013 announcement in Nature relating how they created a particularly pathogenic human-targeting coronavirus by inserting four bat coronaviruses into human cells containing the ACE2 receptor.
"Bat Woman" Shi Zhengli releases a bat
2013
In October 2013, the U.S. government stops all federal funding for “Gain-of-Function” studies. Particular concerns are raised about making influenza, SARS, and Middle East respiratory syndrome (MERS) “even more infectious and lethal.”
“The only impact of this work is the creation, in a lab, of a new, non-natural risk,” charges Richard Ebright, a molecular biologist and biodefence expert at Rutgers University, after more than 200 scientists call for the gain-of-function lab experiments to be halted.
As head of NIAID since 1984, Dr. Anthony Fauci continues to promote Gain-of-Function research on bird-flu viruses. Fauci argues that the risking the entire globe is worth investigating possible vaccines for novel pandemics that could only originate in a lab experiment!
Also that year, the Shi lab at the Wuhan Institute of Virology “passages” a bat coronavirus into monkey cells – then tests its infectivity in human cell lines genetically modified for the ACE2 receptor.
“Take a bat coronavirus that is not infectious to humans, and force its selection by culturing it with cells that express human ACE2 receptor,” Nikolai Petrovsky postulates, “and you can force the bat virus to adapt to infect human cells via mutations in its spike protein, which would have the effect of increasing the strength of its binding to human ACE2...”
“The result of these experiments is a virus that is highly virulent in humans but is sufficiently different that it no longer resembles the original bat virus,” explains this Flinders U. professor, who has spent more than two decades attempting to develop vaccines against influenza, Ebola, and SARS. “Because the mutations are acquired randomly by selection there is no signature of a human gene jockey, but this is clearly a virus still created by human intervention” to infect an adapted new human host.
Just before the U.S. Gain of Function research ban goes into effect, Shi co-authors a paper with Ralph Baric at the North Carolina lab excitedly describing “notable pathogenesis” in infected mice – which share many genetic characteristics with humans.
COVID’S LONG MARCH: 2014 - 2016
